Independent · non-commercial · publishes on a quarterly cycle|Current cycle 2026 Q3
Compound Evidence InstituteEvidence synthesis · established 2023Graded assessments of compounds, trials, methods and supply
Document set current to 30 July 2026
Compound monograph · §4

5-amino-1MQ — safety

Adverse events as reported, contraindications, warnings, and an explicit statement of what the safety evidence does not establish.

Document identifier
CEI-MN-047/4
Series
Compound monograph
Version
4.0
Published
07 Jul 2025
Last reviewed
07 Dec 2025
Next review
07 Dec 2027
Identifier
10.71829/cei.mono.47
Certainty
Very low
Cycle
2025 Q3

§4Safety

§4.1Adverse events reported in controlled trials

Table 4. Adverse events for 5-amino-1MQ as reported in the trial safety tables the Institute holds. Percentages are of randomised participants in the stated trial and arm.

EventActive, %Comparator, %Difference, ppSource
No human safety data of any kindNone exists. This compound has not entered clinical development
Iodide loadWhere supplied as the iodide salt, repeated dosing delivers a substantial iodide load with potential thyroid consequences that no study has examined
A difference column is computed by the Institute from the two reported percentages and is not itself a published figure. Confidence intervals for adverse-event differences are not reported in the contributing trials and are not constructed here.

§4.2Contraindications

  • Not established — no human data

§4.3Warnings and precautions

  • No human study of any kind has been conducted
  • The iodide counter-ion delivers an iodide load that may be pharmacologically relevant in its own right and is not accounted for in any dosing information
  • Marketed as a "peptide" by some suppliers; it is a small molecule and the analytical framework differs entirely

§4.4What this section does not establish

Related assessments: tolerability of incretin receptor agonists · acute pancreatitis · the rodent thyroid C-cell signal.[1,2]

References cited on this page

References are numbered in order of first citation in this document. Each superscript in the text links to its entry below.

  1. Lee C, Zeng J, Drew BG, Sallam T, Martin-Montalvo A, Wan J, Kim SJ, Mehta H, Hevener AL, de Cabo R, Cohen P. The mitochondrial-derived peptide MOTS-c promotes metabolic homeostasis and reduces obesity and insulin resistance. Cell Metabolism 2015;21(3):443–454. doi:10.1016/j.cmet.2015.02.009 · PMID 25738459
  2. International Council for Harmonisation of Technical Requirements for Pharmaceuticals for Human Use. ICH Q3C(R9) Impurities: Guideline for Residual Solvents. ICH Harmonised Guideline 2024;Step 4 version. identifier not held by the Institute

Identifiers are reproduced only where the Institute holds them. Where a digital object identifier or PubMed identifier is not shown, the Institute has recorded the journal and year and has not constructed an identifier.

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